File:NDL1075040 臨牀医学講座 第43輯.pdf

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Original file (5,406 × 3,762 pixels, file size: 21.14 MB, MIME type: application/pdf, 44 pages)

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臨牀医学講座   (Wikidata search (Cirrus search) Wikidata query (SPARQL)  Create new Wikidata item based on this file)
Author
加藤, 豊治郎, 1882-1967
image of artwork listed in title parameter on this page
Title
臨牀医学講座
Volume 第43輯
Publisher
金原商店
Language jpn
Publication date

1936

昭11
Place of publication JP
Source

doi:10.11501/1075040

institution QS:P195,Q477675
Alternative Title
InfoField
高血圧の成因と其の療法
Part Title
InfoField
高血圧の成因と其の療法
Part Creator
InfoField
加藤豊治郎 著
Publication Place
InfoField
東京
Subject: NDC
InfoField
492
Extent
InfoField
61p ; 19cm
Material Type
InfoField
Book
Source Identifier: JPNO
InfoField
46074947
Date Digitized: W3CDTF
InfoField
2010-03-31
Audience
InfoField
一般
Title Transcription
InfoField
リンショウ イガク コウザ
Alternative Transcription
InfoField
コウケツアツ ノ セイイン ト ソノ リョウホウ
Volume Transcription
InfoField
43
Publisher Transcription
InfoField
カネハラ ショウテン
Source Identifier: NDLBibID
InfoField
000000735183
Call Number
InfoField
60-1364
Creator Transcription: NDLNA
InfoField
カトウ, トヨジロウ
Creator: NDLNAId
InfoField
00028159
Available: internet
InfoField
2018-02-01
Contents
InfoField
標題

目次

高血壓の成因/1

一、 血壓上昇の基因/1

細小動脈の器質的變化/3

細小動脈の機能的攣縮/5

血管系の神經支配及び血壓調制機/5

血管運動神經系の興奮の原因/7

二、 高血壓を伴ふ疾患/8

三、 高血壓の分類/9

腎性(蒼白)高血壓症と本態的(赤色)高血壓症/9

高血壓症の血管の組織學的變化/11

四、 腎臟の病變と高血壓との關係/13

五、 血壓上昇の機制/16

腎性高血壓症の基因/17

アドレナリン/18

交感神經の興奮性を亢進せしむる物質/19

血管收縮性物質/21

血壓上昇に關する物質の發生の機制/22

代償現象としての高血壓/25

本態的高血壓症の基因/26

物理學的基因/27

植物性神經ノイローゼ/29

神經中樞の機能異常/30

中樞の酸素供給不足/31

腦内壓の亢進/33

内分泌異常/34

副腎髓質/35

腦下垂體後葉及び前葉/36

甲状腺、生殖腺/38

高血壓の治療/39

一、 血壓を下降せしむる事の可否/39

血壓降下の不利/39

血壓降下の有利/41

血壓降下の注意/45

二、 高血壓患者の攝生/46

安靜

睡眠

食事

飮料

嗜好品

三、 血壓降下劑/51

プリン誘導體

亞硝酸鹽

パパヴエリン

アミン

燐酸鹽

鎭靜劑

催眠劑

鎭痛劑

酒精屬の麻醉劑

植物性神經毒

阿片

ヂギタリス

沃度

弗化水素

ロダン

高張葡萄糖溶液

臟器製劑

内分泌劑

四、 物理的療法/58

瀉血

光線浴

温浴

酸素吸入

陰イオン吸入

ラヂウム

レントゲン

デアテルミー

短波

超短波

五、 外科的療法/61

Licensing

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Public domain
According to Japanese Copyright Law (June 1, 2018 grant) the copyright on this work has expired and is as such public domain. According to articles 51, 52, 53 and 57 of the copyright laws of Japan, under the jurisdiction of the Government of Japan works enter the public domain 50 years after the death of the creator (there being multiple creators, the creator who dies last) or 50 years after publication for anonymous or pseudonymous authors or for works whose copyright holder is an organization.

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